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Dynamics of a nonlocal epidemic model with a new free boundary condition, part 1: Spreading-vanishing dichotomy

Yao Chen, Yihong Du, Wan-Tong Li, Rong Wang

TL;DR

The paper studies a nonlocal Fisher-KPP epidemic model on a dynamically expanding interval with a new free boundary condition linking boundary motion to the weighted internal population and outward flux. It proves global existence and uniqueness, develops comparison principles, and analyzes a principal eigenvalue framework to characterize the spreading-vanishing dichotomy. The work derives sharp criteria for spreading vs. vanishing based on the basic reproduction number $\\mathcal{R}_0 = \\frac{e G'(0)}{ab}$, the initial habitat size, diffusion rates, and kernel properties, including a diffusion-rate threshold $d^*$ and an initial-size threshold $L^*$. This Part 1 lays the groundwork for Part 2, which will determine the spreading speed and rates for various kernel classes. $

Abstract

This paper investigates the long-time dynamics of a nonlocal epidemic model with free boundaries, where a pathogen with density $u(t,x)$ and the infected humans with density $v(t,x)$ evolve according to a reaction-diffusion system with nonlocal diffusion over a one dimensional interval $[g(t), h(t)]$, which represents the epidemic region expanding through its boundaries $x=g(t)$ and $x=h(t)$, known as free boundaries. Such a model with free boundary conditions based on those of Cao et al. \cite{fb27} was considered by several works. Inspired by recent works of Feng et al. \cite{fb20} and Long et al. \cite{fb5}, we propose a new free boundary condition, where the expansion rate of the epidemic region, determined by $h'(t)$ and $g'(t)$, is proportional to a linear combination of the outward flux of the pathogen \(u\) through the range boundary (as in \cite{fb27}) and the weighted total population of infected individuals \(v\) within the region (as in \cite{fb5}). We prove that the system under this new free boundary condition is well-posed, and its long-time dynamical behavior is characterized by a spreading-vanishing dichotomy. Moreover, we obtain sharp criteria for this dichotomy, including a sharp threshold in terms of the initial data $(u_0,v_0)$; and by studying a related eigenvalue problem, we also find a sharp threshold in terms of the diffusion rate, which complements related results in Nguyen and Vo \cite{fb7}. This is Part $1$ of a two part series. In Part $2$, we will determine the spreading speed of the model when spreading occurs, and for some typical classes of kernel functions, we will obtain the precise rates of accelerated spreading.

Dynamics of a nonlocal epidemic model with a new free boundary condition, part 1: Spreading-vanishing dichotomy

TL;DR

The paper studies a nonlocal Fisher-KPP epidemic model on a dynamically expanding interval with a new free boundary condition linking boundary motion to the weighted internal population and outward flux. It proves global existence and uniqueness, develops comparison principles, and analyzes a principal eigenvalue framework to characterize the spreading-vanishing dichotomy. The work derives sharp criteria for spreading vs. vanishing based on the basic reproduction number , the initial habitat size, diffusion rates, and kernel properties, including a diffusion-rate threshold and an initial-size threshold . This Part 1 lays the groundwork for Part 2, which will determine the spreading speed and rates for various kernel classes. $

Abstract

This paper investigates the long-time dynamics of a nonlocal epidemic model with free boundaries, where a pathogen with density and the infected humans with density evolve according to a reaction-diffusion system with nonlocal diffusion over a one dimensional interval , which represents the epidemic region expanding through its boundaries and , known as free boundaries. Such a model with free boundary conditions based on those of Cao et al. \cite{fb27} was considered by several works. Inspired by recent works of Feng et al. \cite{fb20} and Long et al. \cite{fb5}, we propose a new free boundary condition, where the expansion rate of the epidemic region, determined by and , is proportional to a linear combination of the outward flux of the pathogen through the range boundary (as in \cite{fb27}) and the weighted total population of infected individuals within the region (as in \cite{fb5}). We prove that the system under this new free boundary condition is well-posed, and its long-time dynamical behavior is characterized by a spreading-vanishing dichotomy. Moreover, we obtain sharp criteria for this dichotomy, including a sharp threshold in terms of the initial data ; and by studying a related eigenvalue problem, we also find a sharp threshold in terms of the diffusion rate, which complements related results in Nguyen and Vo \cite{fb7}. This is Part of a two part series. In Part , we will determine the spreading speed of the model when spreading occurs, and for some typical classes of kernel functions, we will obtain the precise rates of accelerated spreading.
Paper Structure (10 sections, 34 theorems, 190 equations)

This paper contains 10 sections, 34 theorems, 190 equations.

Key Result

Theorem 1.1

$($Global existence and uniqueness$)$. Suppose that (J) and (W) hold, $G(u)$ satisfies (G1)-(G2), and $\left(u_0(x), v_0(x)\right)$ satisfies B. Then problem A admits a unique solution $(u, v, g, h)$ defined for all $t > 0$.

Theorems & Definitions (37)

  • Theorem 1.1
  • Theorem 1.2
  • Theorem 1.3
  • Theorem 1.4
  • Remark 1.5
  • Theorem 1.6
  • Lemma 2.1
  • Lemma 2.2
  • Lemma 2.3
  • Lemma 2.4
  • ...and 27 more