Translation invariant defects as an extension of topological symmetries
Federico Ambrosino, Ingo Runkel, Gérard M. T. Watts
TL;DR
This work extends the conventional topological symmetry of 2d QFT by introducing translation invariant line defects, which admit non-singular fusion and form a monoidal category that contains topological defects as a subcategory. It provides a perturbative description via a chiral TFT off a bulk perturbation, establishing a commutation condition that determines when a defect remains translation invariant to all orders; translation invariant defects, under RG flow, flow to IR topological defects, enriching the detectable symmetry content. The authors construct the perturbative defect category $\mathcal{T}_{pert}$ with a precise fusion rule and derive functional relations in Ising and Lee-Yang models, constraining RG endpoints. They culminate with a categorical formulation in terms of Yetter-Drinfeld modules, showing how translation invariant defects correspond to Hopf-algebraic data and revealing non-semisimple, infinitely rich defect theories in minimal models. Overall, the paper provides both a perturbative mechanism and a rigorous algebraic framework for studying translation invariant defects as an extension of topological symmetries in two-dimensional QFT.
Abstract
The modern way to understand symmetries of a quantum field theory is via its topological defects in various dimensions. In this contribution to the proceedings we focus on line defects in 2d QFT and we point out that topological defects naturally embed into a larger class, namely translation invariant defects. The latter still allow for non-singular fusion and one obtains a monoidal category of translation invariant defects which contains that of topological defects as a full subcategory. We give a simple perturbative description of translation invariant defects in a perturbed conformal field theory via chiral three-dimensional topological field theory. We show in the example of the Ising CFT and the Lee-Yang CFT that even if no topological defects survive the deformation, some translation invariant defects still do.
