Learning Low-Level Causal Relations using a Simulated Robotic Arm
Miroslav Cibula, Matthias Kerzel, Igor Farkaš
TL;DR
This work investigates learning and exploiting low-level causal relations in robotic systems using simulated data. By training forward and inverse models on motor-babbling data from a simulated robotic arm and applying SHAP and PDP-based attribution, the authors extract actionable insights about how individual state features (robot joints and environmental objects) causally affect outcomes, enabling dimensionality reduction and explainability. The forward model supports multi-step mental simulation (up to 10 steps), and two inverse-model architectures (monolithic and pre-computation) offer viable pathways for action prediction, with explainability analyses revealing which features matter for specific tasks. The approach promises more robust, explainable planning and imitation-learning–driven control in complex robotic environments, especially where intuitive physics and low-level causal structure drive behavior.
Abstract
Causal learning allows humans to predict the effect of their actions on the known environment and use this knowledge to plan the execution of more complex actions. Such knowledge also captures the behaviour of the environment and can be used for its analysis and the reasoning behind the behaviour. This type of knowledge is also crucial in the design of intelligent robotic systems with common sense. In this paper, we study causal relations by learning the forward and inverse models based on data generated by a simulated robotic arm involved in two sensorimotor tasks. As a next step, we investigate feature attribution methods for the analysis of the forward model, which reveals the low-level causal effects corresponding to individual features of the state vector related to both the arm joints and the environment features. This type of analysis provides solid ground for dimensionality reduction of the state representations, as well as for the aggregation of knowledge towards the explainability of causal effects at higher levels.
